Apologies here's one link with a few quotes.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5055448/

“In summary, these results indicate that I-BET 525762A-induced mitochondrial respiration is necessary to rescue the bioenergetic defects and maintain survival in human cybrids with mitochondrial CI mutations.”

“A metabolic hallmark of CI malfunction is NADH accumulation caused by reduction in CI-dependent NADH reductase activity… We show that NADH levels are partially restored after bromodomain inhibition and may contribute to the bioenergetic rescue in CI deficient cells.”