This doesn't look good for ACE2 inhibitors and Covid.  Bradykinin causes inflammation in the lungs.  The bolding below is mine.

https://www.healthing.ca/diseases-and-conditions/coronavirus/covid-19-we-may-already-have-treatments-for-lungs-of-jell-o

"What he discovered — and what has put the medical community on notice — was that the lungs of COVID-19 patients expressed enzymes that trigger the production of bradykinin, an inflammatory molecule. Worse, the biological mechanisms that normally keep bradykinin in check become dysregulated due to COVID-19 virus, setting off an inflammatory cascade that scientists believe lead to the cardiovascular and neurological damage in COVID-19 patients, as well as the dramatic loss of lung function.

"Since the start of the pandemic, ACE2 receptors (Angiotensin-Converting Enzyme 2) have been implicated as being the entry points for the virus. ACE2, an enzyme that is found in the lung, kidney, and gastrointestinal tract, has been shown to harbour SARS-CoV-2, the virus that causes COVID-19.

People who are prescribed ACE inhibitors — which are often used to treat conditions like high blood pressure and heart disease — take the medications to relax the blood vessels, lower blood pressure and prevent heart issues. Intriguingly, and supporting Jacobson’s bradykinin theory, many experience a dry cough, lose their sense of smell, headache, and have muscle pain, symptoms that are the hallmarks of a COVID-19 infection.

ACE2 also regulates bradykinin, though in COVID-19 patients, it is “significantly downregulated,” meaning it’s not doing its job. “When you take it away, you’re not regulating bradykinin,” says Jacobson. “You’re starting this whole cascade.”